Rheumatoid Disease

Introduction:

Good morning, to all you visitors, friends and supporters of The Rheumatoid Disease Foundation. I’m honored to have been asked by The Rheumatoid Disease Foundation to speak to you and share with you some of the exciting new developments and advances that are being made concerning the treatment of Rheumatoid Arthritis and other Rheumatoid Diseases. I would like to personally thank each and every one of you who have supported The Rheumatoid
Disease Foundation and want you to know that if it had not been for the personal help and financial support of many of you and thousands of other supporters across the entire United States, our progress would have been very minimal, but thanks to you and all our supporters,
our knowledge and research is moving and progressing at a very rapid rate. I was asked to speak on the anti-amoebic treatment of Rheumatoid Disease and this is a very broad subject. I know that there are some people here who are not familiar with our work so I will give a rapid background review of our work, what has been done, and then go into the actual treatments and support methods being recommended at the present time.

Rheumatoid Diseases;

Now you have heard me refer to the term Rheumatoid Diseases and let me clarify this term. Dr. Wyburn-Mason was able to isolate and identify heavy concentrations of free-living amoebae which he called the Limax Amoebae in the involved tissues of most of the socalled auto-immune or collagen diseases as well as several types of cancers that primarily involved the lymphatic system as lymphomas. Auto-immune diseases are those in which the white blood cells are
tying to fight some agent (such as a germ or chemical) in the tissues and they are not able to tell the difference between the agent and normal tisssues so they attack normal tissues as well as the agent and this results in severe tissue destruction in the tissues involved. Dr. Wyburn-Mason found the amoebae in all these tissues and learned that the amoebae can attack any tissue in the body. Rheumatoid Disease is not simply a disease of the joints but a generalized condition and every tissue of the body at some time has been reported to be affected. The same tissue changes seen under a microscope as are found in the joint tissues can be seen in other body tissues and consist of invasion by certain white blood cells as lymphocytes and plasmacytes along with inflamed small arteries. If they attack the joints, the disease is called Rheumatoid Arthritis. If they attack the colon, the condition is called Ulcerative Colitis. Small intestine, Crohn’s Disease; arteries: Periarteritis Nodosum; blood: [hemolytic disease]; [connective tissue, skin, organs]: Lupus Erythematosis; thyroid: Hasimoto’s Thyroiditis; nerves: Multiple Sclerosis; salivary glands: Sjorgrens Syndrome; muscle: Dermatomyositis; skin: Psoriasis or Scleroderma.
These are just a few of the various diseases that Dr. Wyburn- Mason has been able to isolate the amoebae from. He therefore calls these diseases where the limax amoebae are found “the Rheumatoid Diseases” and many of these diseases have gone into remission after being treated by anti-amoebic medications. As examples, I have had about 2 out 3 Psoriasis and Lupus Erythematosis patients go into remission after trying the anti-amoebic treatment. I’ve had about 50% of Ulcerative Colitis or Crohn’s Disease patients go into remission. Of the Rheumatoid Arthritis patients treated with various antiamoebic medications, I have found about 80%, 8 out of 10 patients are very significantly relieved or they go into remission. Dr. Robert Bingham of Desert Hot Springs, California, has treated hundreds of Rheumatoid Arthritis patients and his results are very close to my own. Dr. Paul Pybus, The Foundation’s Chief Medical Advisor,
also has had results that fairly closely parallel those of my own. Some physicians have gotten even better results and some have even reported poor results. We haven’t determined as yet why this is so, but we are working on those factors that play a part in influencing treatment medications.
1. Methods of administering the anti-amoebic medications.
2. The diet followed by the patients and nutritional supplements provided.
3. The amounts and types of exercise recommended.
4. The mental attitude and hope instilled into the patients by various physicians.
5. The geographic areas of the country involved.
6. Possibly other types of germs that may be involved or different species of the amoebae that may be resistant to the present available medications.
7. The presence of allergies or co-existing infections that play a part in weakening the immune system.
8. Digestive disturbances and faulty absorption of necessary nutrients, foods and supplements. Candida albicans. Anyhow, several physicians are working on these factors, and we believe with time that we will be able to solve to a great extent the unanswered questions that remain. We are tremendously excited and enthused about our progress so far and are anxiously awaiting the
results of presently on-going studies that have been made possible because of the loyal support of the thousands of concerned members of the Rheumatoid Disease Foundation that are helping us.